Beriberi history and symptoms: Difference between revisions
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==Overview== | ==Overview== | ||
Revision as of 16:00, 12 November 2019
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Abdelrahman Ibrahim Abushouk, MD[2]
Overview
History and Symptoms
History of Beriberi
Symptoms of Beriberi
- Cardiovascular or wet beriberi is manifested by cardiac hypertrophy and heart failure with high cardiac output.[1]
- Tachycardia,
- Respiratory distress,
- Edema of the legs
- Severe lactic acidosis.
- Neurological or dry beriberi is manifested by polyneuritis (sometimes paralysis) that typically starts in the lower limbs and progresses to the upper limbs,
- Exaggeration, followed by loss of deep tendon reflexes
- Muscle weakness and muscle pain
- Convulsions.
- Gastrointestinal symptoms are due to delayed gastric emptying and colon dilation.
- Loss of appetite,
- Vague abdominal complaints
- Constipation
- Nausea and vomiting in advanced stages.
- Wernicke-korsakoff syndrome:
- Wernicke's disease is a triad of nystagmus, ophthalmoplegia, and ataxia, along with confusion.
- Korsakoff's psychosis is impaired short-term memory and confabulation with otherwise grossly normal cognition.
- This combination is almost exclusively described in chronic alcoholics with thiamine deficiency.[2]
References
- ↑ Chisolm-Straker M, Cherkas D (2013). "Altered and unstable: wet beriberi, a clinical review". J Emerg Med. 45 (3): 341–4. doi:10.1016/j.jemermed.2013.04.022. PMID 23849362.
- ↑ Shible AA, Ramadurai D, Gergen D, Reynolds PM (2019). "Dry Beriberi Due to Thiamine Deficiency Associated with Peripheral Neuropathy and Wernicke's Encephalopathy Mimicking Guillain-Barré syndrome: A Case Report and Review of the Literature". Am J Case Rep. 20: 330–334. doi:10.12659/AJCR.914051. PMC 6429982. PMID 30862772.