Wolff-Parkinson-White syndrome overview: Difference between revisions
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==Overview== | ==Overview== | ||
[[Wolff-Parkinson-White]] ([[WPW]]) syndrome is the most common cause of [[ventricular pre-excitation]] and the second common cause of [[supraventricular tachycardia]]. There is a [[muscle fiber]] that bridges the [[atrioventricular groove]] providing electrical continuity between the atrium and ventricle in parallel to the [[atrioventricular node-His-Purkinje axis]]. The [[atrial]] impulse activates the entire or part of the [[ventricle]] or the [[ventricular impulse]] activates the entire [[atrium]] or part of it, earlier than normally be expected. Patients with [[WPW syndrome]] may present with abrupt [[palpitation]], [[presyncope]], [[syncope]], or [[ sudden cardiac death]]([[SCD]]). In some patients, [[SCD]] is the first presentation of [[WPW syndrome]], especially in the setting of [[atrial fibrillation]] with a [[rapid ventricular response]]. | [[Wolff-Parkinson-White]] ([[WPW]]) syndrome is the most common cause of [[ventricular pre-excitation]] and the second common cause of [[supraventricular tachycardia]]. There is a [[muscle fiber]] that bridges the [[atrioventricular groove]] providing electrical continuity between the atrium and ventricle in parallel to the [[atrioventricular node-His-Purkinje axis]]. The [[atrial]] impulse activates the entire or part of the [[ventricle]] or the [[ventricular impulse]] activates the entire [[atrium]] or part of it, earlier than normally be expected. Patients with [[WPW syndrome]] may present with abrupt [[palpitation]], [[presyncope]], [[syncope]], or [[ sudden cardiac death]]([[SCD]]). In some patients, [[SCD]] is the first presentation of [[WPW syndrome]], especially in the setting of [[atrial fibrillation]] with a [[rapid ventricular response]]. [[Wolff-Parkinson-White syndrome]] is named after the cardiologists [[Louis Wolff]], John Parkinson, and [[Paul Dudley White]] who gave a definitive description of the conduction disorder of the heart in 1930. The term [[Wolff-Parkinson-White syndrome]] was coined in 1940. [[Bundle of Kent]] was first discovered by [[Albert Frank Stanley Kent]], a British physiologist following finding the lateral branch in the atrioventricular groove of the monkey heart. | ||
==Historical Perspective== | ==Historical Perspective== | ||
Revision as of 19:33, 9 November 2020
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Wolff-Parkinson-White syndrome Microchapters |
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Differentiating Wolff-Parkinson-White syndrome from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Wolff-Parkinson-White syndrome overview On the Web |
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Risk calculators and risk factors for Wolff-Parkinson-White syndrome overview |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Sara Zand, M.D.[2] Cafer Zorkun, M.D., Ph.D. [3]
Overview
Wolff-Parkinson-White (WPW) syndrome is the most common cause of ventricular pre-excitation and the second common cause of supraventricular tachycardia. There is a muscle fiber that bridges the atrioventricular groove providing electrical continuity between the atrium and ventricle in parallel to the atrioventricular node-His-Purkinje axis. The atrial impulse activates the entire or part of the ventricle or the ventricular impulse activates the entire atrium or part of it, earlier than normally be expected. Patients with WPW syndrome may present with abrupt palpitation, presyncope, syncope, or sudden cardiac death(SCD). In some patients, SCD is the first presentation of WPW syndrome, especially in the setting of atrial fibrillation with a rapid ventricular response. Wolff-Parkinson-White syndrome is named after the cardiologists Louis Wolff, John Parkinson, and Paul Dudley White who gave a definitive description of the conduction disorder of the heart in 1930. The term Wolff-Parkinson-White syndrome was coined in 1940. Bundle of Kent was first discovered by Albert Frank Stanley Kent, a British physiologist following finding the lateral branch in the atrioventricular groove of the monkey heart.