Acute disseminated encephalomyelitis CT: Difference between revisions
| Line 3: | Line 3: | ||
{{CMG}}; {{AE}} {{Sujaya}} | {{CMG}}; {{AE}} {{Sujaya}} | ||
==Overview== | ==Overview== | ||
The appearance of [[ADEM]] on [[cranial]] [[CT]] has rarely been reported. There is a delay between the onset of the [[clinical signs]] and the appearance of [[lesions]] on [[CT scan]]. The correlation was limited between the [[clinical]] course and the [[anatomical]] distribution and type of abnormality seen on [[CT scan]]. | |||
==CT== | ==CT== | ||
Latest revision as of 07:51, 19 November 2022
|
Acute disseminated encephalomyelitis Microchapters |
|
Differentiating Acute disseminated encephalomyelitis from other Diseases |
|---|
|
Diagnosis |
|
Treatment |
|
Case Studies |
|
Acute disseminated encephalomyelitis CT On the Web |
|
American Roentgen Ray Society Images of Acute disseminated encephalomyelitis CT |
|
Directions to Hospitals Treating Acute disseminated encephalomyelitis |
|
Risk calculators and risk factors for Acute disseminated encephalomyelitis CT |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sujaya Chattopadhyay, M.D.[2]
Overview
The appearance of ADEM on cranial CT has rarely been reported. There is a delay between the onset of the clinical signs and the appearance of lesions on CT scan. The correlation was limited between the clinical course and the anatomical distribution and type of abnormality seen on CT scan.
CT
The appearance of ADEM on cranial CT has rarely been reported. There is a delay between the onset of the clinical signs and the appearance of lesions on CT scan. The correlation was limited between the clinical course and the anatomical distribution and type of abnormality seen on CT scan[1]. The findings that have been elucidated in some case reports are[2]:
- Hypodense lesions in the white matter, basal ganglia and brainstem edema
- Contrast-induced spotty, nodular or gyral enhancement
- The enhancement resolved with steroids, leaving persistent hypoattenuated areas, indicating dual components in the pathogenesis of ADEM. Thr vasculitic component is steroid- responsive while the demyelinating component is not.
References
- ↑ Lukes SA, Norman D (1983). "Computed tomography in acute disseminated encephalomyelitis". Ann Neurol. 13 (5): 567–72. doi:10.1002/ana.410130516. PMID 6870208.
- ↑ Thajeb P, Chen ST (1989). "Cranial computed tomography in acute disseminated encephalomyelitis". Neuroradiology. 31 (1): 8–12. doi:10.1007/BF00342021. PMID 2717010.