Hyperparathyroidism pathophysiology: Difference between revisions
Created page with "__NOTOC__ {{Hyperparathyroidism}} ==Overview== ==Pathohysiology== ===Primary Hyperparathyroidism=== {{main|Primary hyperparathyroidism}} * The most common cause is a benign p..." |
No edit summary |
||
Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
{{Hyperparathyroidism}} | {{Hyperparathyroidism}} | ||
{{CMG}} | |||
==Overview== | ==Overview== |
Revision as of 14:05, 20 September 2012
Hyperparathyroidism Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Hyperparathyroidism pathophysiology On the Web |
American Roentgen Ray Society Images of Hyperparathyroidism pathophysiology |
Risk calculators and risk factors for Hyperparathyroidism pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathohysiology
Primary Hyperparathyroidism
- The most common cause is a benign parathyroid adenoma that loses its sensitivity to circulating calcium levels. Usually, only one of the four parathyroid glands is affected.
- A less common cause is from multiple endocrine neoplasia (MEN).
Secondary Hyperparathyroidism
Secondary hyperparathyroidism is due to resistance to the actions of PTH, usually due to chronic renal failure. The bone disease in secondary parathyroidism along with renal failure is termed renal osteodystrophy.
Tertiary Hyperparathyroidism
Tertiary hyperparathyroidism, quartary and quintary hyperparathyroidism are rare forms that are caused by long lasting disorders of the calcium feedback control system. When the hyperparathyroidism can not be corrected by medication one calls it tertiary hyperparathyroidism.