Cryptosporidiosis causes: Difference between revisions

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==Overview==
==Overview==
==Causes==
[[Cryptosporidium]] is a genus of [[protozoan]] parasites within the phylum Apicomplexa, and its taxonomy continues to evolve. Cryptosporidiosis is a zoonotic disease that causes clinical disease in both humans and animals; species names are based primarily on the animal species serving as host . Revised Cryptosporidium taxonomy based on recent advances in molecular laboratory testing methods has clarified that multiple species infect humans. C. hominis (known previously as C. parvum genotype I) primarily infects humans and exists in a human-to-human transmission cycle. C. parvum (known previously as C. parvum genotype II) can infect both humans and ruminants, each with their own transmission cycles that intersect in zoonotic disease. In addition, molecular studies have demonstrated that humans are susceptible to infection with multiple subtypes of C. parvum and C. hominis . To a lesser extent, human infections also have been documented with C. felis, from cats; C. canis, from dogs; C. meleagridis, from birds; C. suis, from pigs; C. muris, from rodents; and a C. cervine genotype from various animals . Illnesses caused by infection with the different Cryptosporidium species might differ clinically  
Cryptosporidium is a genus of protozoan parasites within the phylum Apicomplexa, and its taxonomy continues to evolve. Cryptosporidiosis is a zoonotic disease that causes clinical disease in both humans and animals; species names are based primarily on the animal species serving as host . Revised Cryptosporidium taxonomy based on recent advances in molecular laboratory testing methods has clarified that multiple species infect humans. C. hominis (known previously as C. parvum genotype I) primarily infects humans and exists in a human-to-human transmission cycle. C. parvum (known previously as C. parvum genotype II) can infect both humans and ruminants, each with their own transmission cycles that intersect in zoonotic disease. In addition, molecular studies have demonstrated that humans are susceptible to infection with multiple subtypes of C. parvum and C. hominis . To a lesser extent, human infections also have been documented with C. felis, from cats; C. canis, from dogs; C. meleagridis, from birds; C. suis, from pigs; C. muris, from rodents; and a C. cervine genotype from various animals . Illnesses caused by infection with the different Cryptosporidium species might differ clinically  
==References==
==References==
{{Reflist|2}}
{{Reflist|2}}


[[Category:Parasitic diseases]]
[[Category:Parasitic diseases]]
[[Category:Infectious disease]]
[[Category:Infectious disease]]
[[Category:Disease]]
[[Category:Disease]]


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Revision as of 14:30, 21 November 2012

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Cryptosporidiosis Microchapters

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Overview

Cryptosporidium is a genus of protozoan parasites within the phylum Apicomplexa, and its taxonomy continues to evolve. Cryptosporidiosis is a zoonotic disease that causes clinical disease in both humans and animals; species names are based primarily on the animal species serving as host . Revised Cryptosporidium taxonomy based on recent advances in molecular laboratory testing methods has clarified that multiple species infect humans. C. hominis (known previously as C. parvum genotype I) primarily infects humans and exists in a human-to-human transmission cycle. C. parvum (known previously as C. parvum genotype II) can infect both humans and ruminants, each with their own transmission cycles that intersect in zoonotic disease. In addition, molecular studies have demonstrated that humans are susceptible to infection with multiple subtypes of C. parvum and C. hominis . To a lesser extent, human infections also have been documented with C. felis, from cats; C. canis, from dogs; C. meleagridis, from birds; C. suis, from pigs; C. muris, from rodents; and a C. cervine genotype from various animals . Illnesses caused by infection with the different Cryptosporidium species might differ clinically

References


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