Vancomycin-resistant Staphylococcus aureus historical perspective: Difference between revisions
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==Overview== | ==Overview== | ||
VISA (vancomycin-intermediate ''Staphylococcus aureus'') was first identified in Japan in 1996 and has since been found in hospitals in England, France, the U.S., Asia and Brazil. It is also termed GISA (glycopeptide-intermediate ''Staphylococcus aureus'') indicating resistance to all glycopeptide antibiotics. These bacterial strains present a thickening of the cell wall which is believed to deplete the vancomycin available to kill the bacteria. | VISA (vancomycin-intermediate ''[[Staphylococcus aureus]]'') was first identified in Japan in 1996 and has since been found in hospitals in England, France, the U.S., Asia and Brazil. It is also termed GISA (glycopeptide-intermediate ''Staphylococcus aureus'') indicating resistance to all glycopeptide antibiotics. These bacterial strains present a thickening of the cell wall which is believed to deplete the vancomycin available to kill the bacteria. | ||
==References== | ==References== |
Revision as of 13:47, 17 December 2012
Vancomycin-resistant Staphylococcus aureus Microchapters |
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Vancomycin-resistant Staphylococcus aureus historical perspective On the Web |
American Roentgen Ray Society Images of Vancomycin-resistant Staphylococcus aureus historical perspective |
FDA on Vancomycin-resistant Staphylococcus aureus historical perspective |
CDC on Vancomycin-resistant Staphylococcus aureus historical perspective |
Vancomycin-resistant Staphylococcus aureus historical perspective in the news |
Blogs on Vancomycin-resistant Staphylococcus aureus historical perspective |
Directions to Hospitals Treating Vancomycin-resistant Staphylococcus aureus |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
VISA (vancomycin-intermediate Staphylococcus aureus) was first identified in Japan in 1996 and has since been found in hospitals in England, France, the U.S., Asia and Brazil. It is also termed GISA (glycopeptide-intermediate Staphylococcus aureus) indicating resistance to all glycopeptide antibiotics. These bacterial strains present a thickening of the cell wall which is believed to deplete the vancomycin available to kill the bacteria.