Atherosclerosis risk factors: Difference between revisions
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(Created page with "__NOTOC__ {{Atherosclerosis}} {{CMG}} ==Risk Factors== ===Physiologic factors that increase risk=== Various anatomic, physiological & behavioral risk factors for atherosclero...") |
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==Risk Factors== | ==Risk Factors== | ||
===Physiologic factors that increase risk=== | ===Physiologic factors that increase risk=== | ||
Various anatomic, physiological & behavioral risk factors for atherosclerosis are known. These can be divided into various categories: congenital | Various anatomic, physiological & behavioral risk factors for atherosclerosis are known. These can be divided into various categories: congenital vs acquired, modifiable or not, classical or non-classical. The points labelled '+' in the following list form the core components of "[[metabolic syndrome]]": | ||
* [[Senescence|Advanced age]] | * [[Senescence|Advanced age]] | ||
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* [[Tobacco smoking]] | * [[Tobacco smoking]] | ||
* Having [[hypertension|high]] [[blood pressure]] + | * Having [[hypertension|high]] [[blood pressure]] + | ||
* Being [[obesity|obese]] (in particular [[central obesity]], also referred to as | * Being [[obesity|obese]] (in particular [[central obesity]], also referred to as abdominal or male-type obesity) + | ||
* A [[sedentary lifestyle]] | * A [[sedentary lifestyle]] | ||
* Having close relatives who have had some complication of atherosclerosis (eg. [[coronary heart disease]] or [[stroke]]) | * Having close relatives who have had some complication of atherosclerosis (eg. [[coronary heart disease]] or [[stroke]]) |
Revision as of 19:36, 22 February 2013
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Risk Factors
Physiologic factors that increase risk
Various anatomic, physiological & behavioral risk factors for atherosclerosis are known. These can be divided into various categories: congenital vs acquired, modifiable or not, classical or non-classical. The points labelled '+' in the following list form the core components of "metabolic syndrome":
- Advanced age
- Male sex
- Having Diabetes or Impaired glucose tolerance (IGT) +
- Dyslipoproteinemia (unhealthy patterns of serum proteins carrying fats & cholesterol): +
- High serum concentration of low density lipoprotein (LDL, "bad if elevated concentrations and small"), Lipoprotein(a) (a variant of LDL), and / or very low density lipoprotein (VLDL) particles, i.e. "lipoprotein subclass analysis"
- Low serum concentration of functioning high density lipoprotein (HDL "protective if large and high enough" particles), i.e. "lipoprotein subclass analysis"
- Tobacco smoking
- Having high blood pressure +
- Being obese (in particular central obesity, also referred to as abdominal or male-type obesity) +
- A sedentary lifestyle
- Having close relatives who have had some complication of atherosclerosis (eg. coronary heart disease or stroke)
- Elevated serum levels of homocysteine
- Elevated serum levels of uric acid (also responsible for gout)
- Elevated serum fibrinogen concentrations +
- Chronic systemic inflammation as reflected by upper normal WBC concentrations, elevated hs-CRP and many other blood chemistry markers, most only research level at present, not clinically done.[1]
- Stress or symptoms of clinical depression
- Hypothyroidism (a slow-acting thyroid)
- High intake of trans-fats and saturated fats in diet
References
- ↑ Deepak L. Bhatt, MD; Eric J. Topol, MD Need to Test the Arterial Inflammation Hypothesis, 2002, referenced on 4/1/06