Pulseless electrical activity causes: Difference between revisions
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===Causes in Alphabetical Order=== | ===Causes in Alphabetical Order=== | ||
===Causes in Alphabetical Order=== | |||
{{col-begin|width=80%}} | {{col-begin|width=80%}} | ||
{{col-break|width=33%}} | {{col-break|width=33%}} | ||
* | *[[Acetylcholine]] | ||
* | *[[Acute coronary syndrome]] | ||
* | *[[Acute rheumatic fever]] | ||
* | *[[Amiodarone]] | ||
* | *[[Andersen cardiodysrhythmic periodic paralysis]] | ||
* | *[[Anthracyclines]] | ||
* | *[[Beta-blockers]] | ||
* | *[[Brugada syndrome]] | ||
* | *[[Calcium channel blockers]] | ||
* | *[[carbamate|Carbamate poisoning]] | ||
* | *[[Cardiac catheterization]] | ||
*[[Cardiac resynchronization therapy]] | |||
*[[Cardiac transplantation]] | |||
*[[Cardiac tumor]] | |||
*[[Cholinesterase inhibitors]] | |||
*[[Complete heart block]] | |||
*[[Congenital heart disease]] | |||
*[[Congestive heart failure]] | |||
*[[Coronary artery bypass grafting]] | |||
*[[Daunorubicin]] | |||
*[[Diabetic ketoacidosis]] | |||
*[[Digitalis]] | |||
*[[Dilated cardiomyopathy]] | |||
*[[Doxorubicin]] | |||
{{col-break|width=33%}} | {{col-break|width=33%}} | ||
* [[ | *[[Edrophonium]] | ||
* [[ | *[[Emery-Dreifuss muscular dystrophy]] | ||
* [[ | *[[Epirubicin]] | ||
* [[ | *[[Heart surgery]] | ||
* [[ | *[[Hyperkalemia]] | ||
* [[ | *[[Hypermagnesemia]] | ||
* [[ | *[[Hypertensive heart disease]] | ||
* [[ | *[[Hyperthyroidism]] | ||
* [[ | *[[Hypertrophic cardiomyopathy]] | ||
* [[ | *[[Hypertrophic cardiomyopathy alcohol septal ablation]] | ||
* [[ | *[[Hypocalcemia]] | ||
* [[ | *[[Hypothermia]] | ||
*[[Hypoxia]] | |||
*[[Idarubicin]] | |||
*[[regional anesthesia|Infraclavicular brachial plexus block]] | |||
*[[Ischemic heart disease]] | |||
*[[Jervell and Lange-Nielsen syndrome]] | |||
*[[Kearns-Sayre syndrome]] | |||
*[[Lev's disease]] | |||
*[[Limb-girdle muscular dystrophy|Limb-girdle muscular dystrophy type 1B (LGMD1B)]] | |||
*[[Long QT syndrome]] | |||
*[[Muscular dystrophy]] | |||
*[[Myocardial bridging]] | |||
*[[Myocardial contusion]] | |||
{{col-break|width=33%}} | {{col-break|width=33%}} | ||
* | *[[Myocardial infarction]] | ||
*[[Myocarditis]] | |||
*[[Myotonic dystrophy]] | |||
*[[Neonatal lupus erythematosus]] | |||
*[[Neostigmine]] | |||
*[[organophosphate|Organophosphate poisoning]] | |||
*[[parathion|Parathion poisoning]] | |||
*[[Pericarditis]] | |||
*[[Procainamide]] | |||
*[[hypothyroidism|Profound hypothyroidism]] | |||
*[[Propafenone]] | |||
*[[Propofol]] | |||
*[[Pyridostigmine]] | |||
*[[Quinidine]] | |||
*[[Renal failure]] | |||
*[[Romano-Ward syndrome]] | |||
*[[anorexia nervosa|Severe anorexia nervosa]] | |||
*[[brain damage|Severe brain injury]] | |||
*[[Takotsubo cardiomyopathy]] | |||
*[[Timothy syndrome]] | |||
*[[Tramadol]] | |||
*[[Valvular heart disease]] | |||
{{col-end}} | {{col-end}} | ||
Revision as of 15:25, 12 September 2013
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Resident Survival Guide |
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Pulseless electrical activity Microchapters |
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Differentiating Pulseless Electrical Activity from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Pulseless electrical activity causes On the Web |
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American Roentgen Ray Society Images of Pulseless electrical activity causes |
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Directions to Hospitals Treating Pulseless electrical activity |
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Risk calculators and risk factors for Pulseless electrical activity causes |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pulseless electrical activity is the initial presenting rhythm in 19.6% of patients who are monitored at the onset of cardiac arrest and 16.5% of patients who present to a prehospital system in full cardiac arrest. [1]
Causes
Life Threatening Causes
Pulseless electrical activity is a life-threatening condition and must be treated as such irrespective of the causes. Life-threatening conditions can result in death or permanent disability within 24 hours if left untreated.
Common Causes
Common causes of PEA include preceding respiratory failure in 40% to 50% of cases, and hypovolemia. The common causes of PEA can be remembered using the mnemonic "The Hs and Ts".[2][3][4]
- Hypovolemia
- Hypoxia
- Hydrogen ions (Acidosis)
- Hypothermia
- Hyperkalemia or Hypokalemia
- Hypoglycemia
- Tablets or Toxins (Drug overdose) such as beta blockers, tricyclic antidepressants, or calcium channel blockers
- Tamponade
- Tension pneumothorax
- Thrombosis (Myocardial infarction)
- Thrombosis (Pulmonary embolism)
- Trauma (Hypovolemia from blood loss)
As noted by repeated balloon inflations in the cardiac catheterization laboratory, transient occlusion of the coronary artery does not cause PEA.
Causes by Organ System
Causes in Alphabetical Order
Causes in Alphabetical Order
References
- ↑ Stueven H, Troiano P, Thompson B, Mateer JR, Kastenson EH, Tonsfeldt D; et al. (1986). "Bystander/first responder CPR: ten years experience in a paramedic system". Ann Emerg Med. 15 (6): 707–10. PMID 3706861.
- ↑ ACLS: Principles and Practice. p. 71-87. Dallas: American Heart Association, 2003. ISBN 0-87493-341-2.
- ↑ ACLS for Experienced Providers. p. 3-5. Dallas: American Heart Association, 2003. ISBN 0-87493-424-9.
- ↑ "2005 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care - Part 7.2: Management of Cardiac Arrest." Circulation 2005; 112: IV-58 - IV-66.