Gliomatosis cerebri other imaging findings
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sujit Routray, M.D. [2]
Overview
Other imaging studies for gliomatosis cerebri include MR spectroscopy (elevated choline/creatine ratio, choline/NAA ratio, and myoinositol), MR perfusion (low/normal relative cerebral blood flow), PET scan (markedly decreased accumulation of [18F]-fluorodeoxyglucose on F-18 FDG PET images, hypermetabolism on C-11 methionine PET images, and marked increase in cerebral blood flow on 15(O)-water PET images), and bone scan (metastasis to bones).[1]
Other Imaging Findings
Other imaging studies for gliomatosis cerebri include MR spectroscopy (elevated choline/creatine ratio, choline/NAA ratio, and myoinositol), MR perfusion (low/normal relative cerebral blood flow), PET scan (markedly decreased accumulation of [18F]-fluorodeoxyglucose on F-18 FDG PET images, hypermetabolism on C-11 methionine PET images, and marked increase in cerebral blood flow on 15(O)-water PET images), and bone scan (metastasis to bones).[1]
1. MR Spectroscopy
Other imaging studies for gliomatosis cerebri include MR spectroscopy. Common findings are described below:[1]
- Elevated choline/creatine ratio
- Elevated choline/NAA ratio
- Marked elevation of myoinositol
2. MR Perfusion
Other imaging studies for gliomatosis cerebri include MR perfusion. Common findings are described below:[1]
- Low/normal relative cerebral blood flow (no vascular hyperplasia)
3. PET
Other imaging studies for gliomatosis cerebri include PET scan. Common features are described below:[1]
- F-18 FDG PET images show markedly decreased accumulation of [18F]-fluorodeoxyglucose (glucose hypometabolism).
- C-11 methionine PET images show hypermetabolism.
- 15(O)-water PET images show a marked increase in cerebral blood flow in gliomatosis cerebri lesions.
4. Bone Scan
Bone scan may be performed to detect metastases of gliomatosis cerebri to bones.
References