Dextro-transposition of the great arteries physical examination

Jump to navigation Jump to search

Dextro-transposition of the great arteries/complete transposition of the great arteries Microchapters

Home

Patient Info

Overview

Pathophysiology

Epidemiology & Demographics

Screening

Natural History, Complications & Prognosis

Causes of dextro-transposition of the great arteries

Differentiating dextro-transposition of the great arteries from other Diseases

Diagnosis

History & Symptoms

Physical Examination

Lab Tests

Electrocardiogram

Chest X Ray

Echocardiography or Ultrasound

Cardiac catheterization

Treatment overview

Medical Therapy

Transposition of the great arteries

Transposition of the great arteries

Transposition of the great arteries


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Associate Editors-In-Chief: Keri Shafer, M.D. [2]; Atif Mohammad, M.D., Priyamvada Singh, MBBS


Physical Examination in Infants with d-TGA

Symmetric cyanosis is the main characteristic in physical appearence of patients with d-TGA, inadequate mixing, and low pulmonary arterial blood flow. Delayed mild cyanosis, and the apearence of congestive heart failure accompanies non-restrictive VSD.

Reversed differential cyanosis (feet less cyanotic than hands) can be a manifestation in patients with d-TGA and large patent ductus arteriosus that has develop early pulmonary vascular disease, reversing the ductal flow if the patient has survived. At this poin in time, the pulmonary arterial blood of high oxygen content enters the aorta and is selectively distribuited to the lower extremities.

The murmur of a large PDA in d-TGA, is usually systolic, seldom continuos, due to the almost exclusive flow during systole from the aorta to the pulmonary artery.

There is a prominent impulse at the LLSB (the RV which is actually the morphologic LV).

The first heart sound (S1) is normal in intensity and splitting because the PR interval and ventricular activation is normal. Due to the anterior location of the aorta, the second heart sound (S2) is accentuated and is usually single.

Systolic murmus are absent in neonates unless a subpulmonic stenosis is present. Short midsystolic murmur originate in the anterior aorta when hypervolemia is present. When the pulmonary vascular resistence is low, a midsystolic murmur is originated in the posterior pulmonary artery, but the murmur is dump by the anterior aorta.

A VSD murmur (holosystolic)is absent at birth, until the pulmonary vascular resistence fall. A subsequent increase in pulmonary resistence shortens and later abolishes the murmur.

References

Acknowledgements and Initial Contributors to Page

Leida Perez, M.D.

External links

nl:Transpositie van de grote vaten

Template:WH Template:WS