Acrodermatitis chronica atrophicans pathophysiology
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Raviteja Guddeti, M.B.B.S. [2]
Acrodermatitis chronica atrophicans Microchapters |
Differentiating Acrodermatitis chronica atrophicans from other Diseases |
---|
Diagnosis |
Treatment |
Case Studies |
Acrodermatitis chronica atrophicans pathophysiology On the Web |
American Roentgen Ray Society Images of Acrodermatitis chronica atrophicans pathophysiology |
Acrodermatitis chronica atrophicans pathophysiology in the news |
Blogs on Acrodermatitis chronica atrophicans pathophysiology |
Risk calculators and risk factors for Acrodermatitis chronica atrophicans pathophysiology |
Overview
This progressive skin process is due to the effect of continuing active infection with the spirochete Borrelia afzelii. B afzelii is the predominant pathophysiology, but may not be the exclusive, etiologic agent of ACA. Borrelia garinii, has also been detected.
Microscopic Pathology
- Light and electron microscopic study of the skin biopsy shows degeneration of the elastica and collagen fibers.[1]