Oesophagostomum pathophysiology
|
Oesophagostomum Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Oesophagostomum pathophysiology On the Web |
|
American Roentgen Ray Society Images of Oesophagostomum pathophysiology |
|
Risk calculators and risk factors for Oesophagostomum pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Please help WikiDoc by adding more content here. It's easy! Click here to learn about editing.
Pathophysiology
In oesophagostomiasis, larvae can invade the colon wall, potentially causing two pervading types of nodular pathology. Multinodular disease is characterized by the formation of many tiny nodular lesions containing worms and pus along the colon wall. About 15% of patients have this form of oesophagostomiasis.[1] Nodules themselves are usually not a problem, but they can give rise to further complications, such as bowel obstruction, peritonitis and intestinal volvulus. In rare cases serious disease can occur including emaciation, fluid in the pericardium, cardiomegaly, hepatosplenomegaly, perisplenitis, and enlargement of the appendix.
Single-nodular disease, more commonly known as Dapaong disease, is characterized by the development of a single mass that develops throughout the colon wall. This is the most common form of oesophagostomiasis, affecting 85% of patients.[2] This nodule can instigate intense tissue reactions that result in the formation of painful projecting masses.
References
- ↑ “GIDEON Infectious Diseases - Diseases.” GIDEON Infectious Disease Database. 5 Feb 2009. <http://web.gideononline.com/web/epidemiology/index.php?gdn_form=ZGlzZWFzZT0xMTY1MA==>.
- ↑ “GIDEON Infectious Diseases - Diseases.” GIDEON Infectious Disease Database. 5 Feb 2009.<http://web.gideononline.com/web/epidemiology/index.php?gdn_form=ZGlzZWFzZT0xMTY1MA==>.