Amnesia differential diagnosis
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Differentiating Amnesia from other Diseases
Differentiating Psychogenic Amnesia from Organic Amnesia
Clinically, psychogenic amnesia is characterized by the loss of the ability to retrieve stored memory without having damages to the brain while organic amnesia is characterized by damages to the medial or anterior temporal and/or prefrontal regions caused by stroke, traumatic brain injury, ischemia, and encephalitis.[1][2] Some characteristics that define organic amnesia is the maintenance of personal identity, basic semantic knowledge and procedural skills as well as neuroradiological images showing cerebral damage to the cortical and/or subcortical areas known to be associated with long-term memory while some characteristics that define psychogenic amnesia is the loss of personal identity, semantic knowledge, and procedural abilities at least in the early phase of amnesia as well as damage directly affecting cerebral areas critical for memory functioning that cannot be detected in clinical history or neuroradiological exams.[2]
Differentiating Disociative Fugue from other Diseases
Some disorders have similar symptoms. The clinician, therefore, in his or her diagnostic attempt has to differentiate against the following disorders which need to be ruled out to establish a precise diagnosis: dementia, head trauma, substance intoxication, early symptoms of neurological disorders (eg multiple sclerosis) may resemble conversion symptoms. Similarly, other psychological disorders may manifest symptoms similar to Dissocative Fugues. These include bipolar disorder, schizophrenia and major depressive disorder.
References
- ↑ Markowitsch HJ (2003). "Psychogenic amnesia". Neuroimage. 20 Suppl 1: S132–8. PMID 14597306.
- ↑ 2.0 2.1 Serra L, Fadda L, Buccione I, Caltagirone C, Carlesimo GA (2007). "Psychogenic and organic amnesia: a multidimensional assessment of clinical, neuroradiological, neuropsychological and psychopathological features". Behav Neurol. 18 (1): 53–64. PMID 17297220.