Renal amyloidosis classification: Difference between revisions

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==Overview==
==Overview==
There is no established system for the classification of [disease name].
Renal amyloidosis can be classified according to the site of amyloid deposition into glomerular, vascular, and tubular amyloidosis.
 
OR
 
[Disease name] may be classified according to [classification method] into [number] subtypes/groups: [group1], [group2], [group3], and [group4].
 
OR
 
[Disease name] may be classified into [large number > 6] subtypes based on [classification method 1], [classification method 2], and [classification method 3].
[Disease name] may be classified into several subtypes based on [classification method 1], [classification method 2], and [classification method 3].
 
OR
 
Based on the duration of symptoms, [disease name] may be classified as either acute or chronic.
 
OR
 
If the staging system involves specific and characteristic findings and features:
According to the [staging system + reference], there are [number] stages of [malignancy name] based on the [finding1], [finding2], and [finding3]. Each stage is assigned a [letter/number1] and a [letter/number2] that designate the [feature1] and [feature2].
 
OR
 
The staging of [malignancy name] is based on the [staging system].
 
OR
 
There is no established system for the staging of [malignancy name].


==Classification==
==Classification==


There is no established system for the classification of [disease name].
Renal amyloidosis may be classified according to site of amyloid deposition into 3 subtypes:<ref name="pmid21360109">{{cite journal |vauthors=Bilginer Y, Akpolat T, Ozen S |title=Renal amyloidosis in children |journal=Pediatr. Nephrol. |volume=26 |issue=8 |pages=1215–27 |date=August 2011 |pmid=21360109 |pmc=3119800 |doi=10.1007/s00467-011-1797-x |url=}}</ref>
 
*Glomerular amyloid deposition (more common and have a poorer prognosis)
OR
*Vascular amyloid deposition
 
*Tubular amyloid deposition
[Disease name] may be classified according to [classification method] into [number] subtypes/groups:  
Renal amyloidosis may be classified according to type of amyloidogenic protein into 3 subtypes:<ref name="pmid23704299">{{cite journal |vauthors=Said SM, Sethi S, Valeri AM, Leung N, Cornell LD, Fidler ME, Herrera Hernandez L, Vrana JA, Theis JD, Quint PS, Dogan A, Nasr SH |title=Renal amyloidosis: origin and clinicopathologic correlations of 474 recent cases |journal=Clin J Am Soc Nephrol |volume=8 |issue=9 |pages=1515–23 |date=September 2013 |pmid=23704299 |pmc=3805078 |doi=10.2215/CJN.10491012 |url=}}</ref>
*[Group1]
{| class="wikitable"
*[Group2]
!
*[Group3]
!Amyloidosis type
*[Group4]
!Amyloidogenic protein
 
|-
OR
| rowspan="4" |Common types
In renal amylodosis, site of amyloid deposition varied in different group of patients. patients having glomerular amyloid deposition are more common and have a poorer prognosis than patients having vascular and tubular amyloid deposition in rheumatoid arthritis-related AA amyloidosis.
| rowspan="3" |AL/AHL/AH
 
(Primary amyloidosis)
[Disease name] may be classified into [large number > 6] subtypes based on:  
|Ig light chains (AL)
*[Classification method 1]
|-
*[Classification method 2]
|Fragments of Ig heavy chains and light chains (AHL)
*[Classification method 3]
|-
 
|Fragments of heavy chains only (AH)
[Disease name] may be classified into several subtypes based on:
|-
*[Classification method 1]
|AA
*[Classification method 2]
(Secondary amyloidosis)
*[Classification method 3]
|Serum amyloid A
 
|-
OR
| rowspan="6" |Rare types
 
|AFib
Based on the duration of symptoms, [disease name] may be classified as either acute or chronic.
|Fibrinogen A α chain
 
|-
OR
|AApo AI/AII/AIV
 
|Apo AI, Apo AII, or Apo AIV
'''If the staging system involves specific and characteristic findings and features:'''
|-
 
|ATTR
According to the [staging system + reference], there are [number] stages of [malignancy name] based on the [finding1], [finding2], and [finding3]. Each stage is assigned a [letter/number1] and a [letter/number2] that designate the [feature1] and [feature2].
|Transthyretin
 
|-
OR
|ALys
 
|Lysozyme
The staging of [malignancy name] is based on the [staging system].
|-
 
|AGel
OR
|Gelsolin
 
|-
There is no established system for the staging of [malignancy name].
|ALECT2
|Leukocyte chemotactic factor 2
|}


==References==
==References==

Latest revision as of 19:50, 17 July 2018

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Shaghayegh Habibi, M.D.[2]

Overview

Renal amyloidosis can be classified according to the site of amyloid deposition into glomerular, vascular, and tubular amyloidosis.

Classification

Renal amyloidosis may be classified according to site of amyloid deposition into 3 subtypes:[1]

  • Glomerular amyloid deposition (more common and have a poorer prognosis)
  • Vascular amyloid deposition
  • Tubular amyloid deposition

Renal amyloidosis may be classified according to type of amyloidogenic protein into 3 subtypes:[2]

Amyloidosis type Amyloidogenic protein
Common types AL/AHL/AH

(Primary amyloidosis)

Ig light chains (AL)
Fragments of Ig heavy chains and light chains (AHL)
Fragments of heavy chains only (AH)
AA

(Secondary amyloidosis)

Serum amyloid A
Rare types AFib Fibrinogen A α chain
AApo AI/AII/AIV Apo AI, Apo AII, or Apo AIV
ATTR Transthyretin
ALys Lysozyme
AGel Gelsolin
ALECT2 Leukocyte chemotactic factor 2

References

  1. ↑ Bilginer Y, Akpolat T, Ozen S (August 2011). "Renal amyloidosis in children". Pediatr. Nephrol. 26 (8): 1215–27. doi:10.1007/s00467-011-1797-x. PMC 3119800. PMID 21360109.
  2. ↑ Said SM, Sethi S, Valeri AM, Leung N, Cornell LD, Fidler ME, Herrera Hernandez L, Vrana JA, Theis JD, Quint PS, Dogan A, Nasr SH (September 2013). "Renal amyloidosis: origin and clinicopathologic correlations of 474 recent cases". Clin J Am Soc Nephrol. 8 (9): 1515–23. doi:10.2215/CJN.10491012. PMC 3805078. PMID 23704299.

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