Dextro-transposition of the great arteries classification: Difference between revisions
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==Classification== | ==Classification== | ||
===Simple d-TGA=== | |||
d-TGA which is not associated with additional cardiac defects. | d-TGA which is not associated with additional cardiac defects. | ||
===Complex d-TGA=== | |||
d-TGA which is associated with other defects. | d-TGA which is associated with other defects. | ||
One of the most common cardiac defects associated with d-TGA is a [[VSD]], which is present in 40% of d-TGA patients. [[Pulmonary stenosis]] and a [[VSD]] are present in 31% of patients. Although it may seem illogical, complex d-TGA presents better chance of survival and less [[child development|developmental]] risks than simple d-TGA, as well as usually requiring fewer invasive[[palliative]] procedures. This is because the [[left-to-right shunt|left-to-right]] and [[bidirectional shunt|bidirectional]] shunting caused by the defects common to complex d-TGA allow a higher amount of oxygen-rich blood to enter the [[systemic circulation]]. However, complex d-TGA may cause a very slight increase to length and risk of the corrective [[surgery]], as most or all other heart defects will normally be repaired at the same time, and the heart becomes "[[irritation|irritated]]" the more it is manipulated. | One of the most common cardiac defects associated with d-TGA is a [[VSD]], which is present in 40% of d-TGA patients. [[Pulmonary stenosis]] and a [[VSD]] are present in 31% of patients. Although it may seem illogical, complex d-TGA presents better chance of survival and less [[child development|developmental]] risks than simple d-TGA, as well as usually requiring fewer invasive[[palliative]] procedures. This is because the [[left-to-right shunt|left-to-right]] and [[bidirectional shunt|bidirectional]] shunting caused by the defects common to complex d-TGA allow a higher amount of oxygen-rich blood to enter the [[systemic circulation]]. However, complex d-TGA may cause a very slight increase to length and risk of the corrective [[surgery]], as most or all other heart defects will normally be repaired at the same time, and the heart becomes "[[irritation|irritated]]" the more it is manipulated. | ||
==References== | ==References== | ||
Latest revision as of 21:16, 7 January 2013
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Dextro-transposition of the great arteries Microchapters |
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Differentiating dextro-transposition of the great arteries from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Dextro-transposition of the great arteries classification On the Web |
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FDA on Dextro-transposition of the great arteries classification |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.B.B.S. [2]; Cafer Zorkun, M.D., Ph.D. [3]; Keri Shafer, M.D. [4]; Assistant Editor(s)-In-Chief: Kristin Feeney, B.S. [5]
Overview
d-TGA is often accompanied by other heart defects, the most common type being intracardiac shunts such as atrial septal defect (ASD) including patent foramen ovale (PFO), ventricular septal defect (VSD), and patent ductus arteriosus (PDA). Stenosis of valves or vessels may also be present.
Classification
Simple d-TGA
d-TGA which is not associated with additional cardiac defects.
Complex d-TGA
d-TGA which is associated with other defects.
One of the most common cardiac defects associated with d-TGA is a VSD, which is present in 40% of d-TGA patients. Pulmonary stenosis and a VSD are present in 31% of patients. Although it may seem illogical, complex d-TGA presents better chance of survival and less developmental risks than simple d-TGA, as well as usually requiring fewer invasivepalliative procedures. This is because the left-to-right and bidirectional shunting caused by the defects common to complex d-TGA allow a higher amount of oxygen-rich blood to enter the systemic circulation. However, complex d-TGA may cause a very slight increase to length and risk of the corrective surgery, as most or all other heart defects will normally be repaired at the same time, and the heart becomes "irritated" the more it is manipulated.
References
Acknowledgements and Initial Contributors to Page
Leida Perez, M.D. nl:Transpositie van de grote vaten Template:WH Template:WS