Postpartum thyroiditis risk factors: Difference between revisions
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**Medications: lithium, amiodarone, interferon alpha, interleukin 2, and the highly active anti-retroviral therapy. | **Medications: lithium, amiodarone, interferon alpha, interleukin 2, and the highly active anti-retroviral therapy. | ||
**Systemic lupus erythromatosis | **Systemic lupus erythromatosis | ||
**Multiple Sclerosis | **[[Multiple Sclerosis]] | ||
**Thyroid hormone resistance syndrome | **[[Thyroid]] hormone resistance syndrome | ||
==References== | ==References== |
Revision as of 14:40, 11 October 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
The most potent risk factor in the development of PPT are genetic and subsequent pregnancies. Other risk factors include smoking, increase or decreased intake of Iodine, hepatitis C, radiations and medications.
Risk Factors
- The most potent risk factor in the development of PPT is genetic. Other risk factors include smoking, increase or decreased intake of Iodine, hepatitis C, radiations and medications.
Common Risk Factors
- Common risk factor in the development of PPT is genetic and includes
- CD60 CTLA-4 gene
- HLA DR3 antigens halpotypes.
- HLA DR4 antigens halpotypes.
- HLA DR5 antigens halpotypes.
- Subsequent pregnancies
Less Common Risk Factors
- Less common risk factors in the development of PPT are environmental and they include:
- Smoking
- Intake of Iodine
- Radiation
- Medications: lithium, amiodarone, interferon alpha, interleukin 2, and the highly active anti-retroviral therapy.
- Systemic lupus erythromatosis
- Multiple Sclerosis
- Thyroid hormone resistance syndrome
References
doi: 10.2478/folmed-2014-0021Template:WH Template:WS