Familial adenomatous polyposis epidemiology and demographics: Difference between revisions
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===Prevalence=== | ===Prevalence=== | ||
*The prevalence of familial adenomatous polyposis is approximately | *The prevalence of familial adenomatous polyposis is approximately 3-10 per 100,000 individuals in the Europe.<ref name="HalfBercovich2009">{{cite journal|last1=Half|first1=Elizabeth|last2=Bercovich|first2=Dani|last3=Rozen|first3=Paul|title=Familial adenomatous polyposis|journal=Orphanet Journal of Rare Diseases|volume=4|issue=1|year=2009|pages=22|issn=1750-1172|doi=10.1186/1750-1172-4-22}}</ref> | ||
*In [year], the incidence/prevalence of [disease name] was estimated to be [number range] cases per 100,000 individuals worldwide. | *In [year], the incidence/prevalence of [disease name] was estimated to be [number range] cases per 100,000 individuals worldwide. | ||
*The prevalence of [disease/malignancy] is estimated to be [number] cases annually. | *The prevalence of [disease/malignancy] is estimated to be [number] cases annually. | ||
===Case-fatality rate/Mortality rate=== | ===Case-fatality rate/Mortality rate=== | ||
*In 1975, the incidence of familial adenomatous polyposis is approximately 13 per 100,000 individuals with a surgical mortality rate of 4.4% in Sweden. | *In 1975, the incidence of familial adenomatous polyposis is approximately 13 per 100,000 individuals with a surgical mortality rate of 4.4% in Sweden.<ref name="pmid">{{cite journal |vauthors=Alm T |title=Surgical treatment of hereditary adenomatosis of the colon and rectum in Sweden during the last 20 years. Part II. Patients with prophylactic operations, primary and late results. Discussion and summary |journal=Acta Chir Scand |volume=141 |issue=3 |pages=228–37 |year=1975 |pmid= |doi= |url=}}</ref> | ||
===Age=== | ===Age=== | ||
*Patients | *Patients in their first and second decades usually develop familial adenomatous polyposis. | ||
*The incidence of [disease name] increases with age; the median age at diagnosis is [#] years. | *The incidence of [disease name] increases with age; the median age at diagnosis is [#] years. | ||
===Race=== | ===Race=== | ||
*There is no racial predilection to [disease name]. | *There is no racial predilection to [disease name]. |
Revision as of 17:07, 26 January 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sadaf Sharfaei M.D.[2], Mohamad Alkateb, MBBCh [3]
Overview
Epidemiology and Demographics
Incidence
- The incidence familial adenomatous polyposis is approximately 12 per 100,000 births in the Europe.[1]
Prevalence
- The prevalence of familial adenomatous polyposis is approximately 3-10 per 100,000 individuals in the Europe.[1]
- In [year], the incidence/prevalence of [disease name] was estimated to be [number range] cases per 100,000 individuals worldwide.
- The prevalence of [disease/malignancy] is estimated to be [number] cases annually.
Case-fatality rate/Mortality rate
- In 1975, the incidence of familial adenomatous polyposis is approximately 13 per 100,000 individuals with a surgical mortality rate of 4.4% in Sweden.[2]
Age
- Patients in their first and second decades usually develop familial adenomatous polyposis.
- The incidence of [disease name] increases with age; the median age at diagnosis is [#] years.
Race
- There is no racial predilection to [disease name].
- [Disease name] usually affects individuals of the [race 1] race. [Race 2] individuals are less likely to develop [disease name].
Gender
- Familial adenomatous polyposis affects men and women equally.[1]
Region
- Familial adenomatous polyposis is a rare disease that is worldwide.
References
- ↑ 1.0 1.1 1.2 Half, Elizabeth; Bercovich, Dani; Rozen, Paul (2009). "Familial adenomatous polyposis". Orphanet Journal of Rare Diseases. 4 (1): 22. doi:10.1186/1750-1172-4-22. ISSN 1750-1172.
- ↑ Alm T (1975). "Surgical treatment of hereditary adenomatosis of the colon and rectum in Sweden during the last 20 years. Part II. Patients with prophylactic operations, primary and late results. Discussion and summary". Acta Chir Scand. 141 (3): 228–37.