Familial adenomatous polyposis medical therapy: Difference between revisions
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==Overview== | ==Overview== | ||
The mainstay of treatment for familial adenomatous polyposis is [[surgery]]. However, [[non-steroidal anti-inflammatory drug]]s ( | The mainstay of treatment for familial adenomatous polyposis is [[surgery]]. However, [[non-steroidal anti-inflammatory drug]]s ([[NSAID]]s) such as [[sulindac]] and [[celecoxib]] are recommended to decrease the size and number of [[colon polyps]]. | ||
==Medical Therapy== | ==Medical Therapy== | ||
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==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Medicine]] | |||
[[Category:Gastroenterology]] | |||
[[Category:Oncology]] | |||
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[[Category:Primary care]] |
Revision as of 14:07, 5 February 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sadaf Sharfaei M.D.[2], Mohamad Alkateb, MBBCh [3]
Overview
The mainstay of treatment for familial adenomatous polyposis is surgery. However, non-steroidal anti-inflammatory drugs (NSAIDs) such as sulindac and celecoxib are recommended to decrease the size and number of colon polyps.
Medical Therapy
- The mainstay of treatment for familial adenomatous polyposis is surgery.
- Pharmacologic medical therapies for familial adenomatous polyposis include non-steroidal anti-inflammatory drugs (NSAIDs).[1][2]
Note (1): non-steroidal anti-inflammatory drugs (NSAIDs) decrease the size and number of colon polyps.
References
- ↑ King JE, Dozois RR, Lindor NM, Ahlquist DA (2000). "Care of patients and their families with familial adenomatous polyposis". Mayo Clin. Proc. 75 (1): 57–67. doi:10.4065/75.1.57. PMID 10630758.
- ↑ Half, Elizabeth; Bercovich, Dani; Rozen, Paul (2009). "Familial adenomatous polyposis". Orphanet Journal of Rare Diseases. 4 (1): 22. doi:10.1186/1750-1172-4-22. ISSN 1750-1172.
- ↑ Amos-Landgraf JM, Kwong LN, Kendziorski CM, Reichelderfer M, Torrealba J, Weichert J, Haag JD, Chen KS, Waller JL, Gould MN, Dove WF (2007). "A target-selected Apc-mutant rat kindred enhances the modeling of familial human colon cancer". Proc. Natl. Acad. Sci. U.S.A. 104 (10): 4036–41. doi:10.1073/pnas.0611690104. PMC 1805486. PMID 17360473.