Aplastic anemia pathophysiology: Difference between revisions
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{{CMG}}; '''Associate Editor(s)-In-Chief:''' [[Priyamvada Singh|Priyamvada Singh, M.D.]] [mailto: | {{CMG}}; '''Associate Editor(s)-In-Chief:''' [[Priyamvada Singh|Priyamvada Singh, M.D.]] [mailto:psingh13579@gmail.com] | ||
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Revision as of 00:38, 2 November 2012
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Aplastic anemia Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Aplastic anemia pathophysiology On the Web |
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American Roentgen Ray Society Images of Aplastic anemia pathophysiology |
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Risk calculators and risk factors for Aplastic anemia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.D. [2]
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Overview
Aplastic anemia is a fatal disorder in which bone marrow's myeloid progenitor cells and stem cells are severely decreased or absent. This may be seen because of either an intrinsic defect of the stem cells or immune-mediated stem cell destruction. As a result of hypocellular stem cells, transfusion-dependent anemia, thrombocytopenia, and severe neutropenia are seen with aplastic anemia. Interferon-activated T lymphocytes are involved in autoimmune destruction of stem cells in both forms of the disease i.e. idiopathic and the acquired. Due to this, immunosuppressive therapy is effective in some patients.