Acute retinal necrosis MRI: Difference between revisions
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==Key MRI Findings for Acute retinal necrosis== | ==Key MRI Findings for Acute retinal necrosis== | ||
MRI imaging may reveal the following indicators of Acute retinal necrosis:<ref name="pmid15569737">{{cite journal |vauthors=Bert RJ, Samawareerwa R, Melhem ER |title=CNS MR and CT findings associated with a clinical presentation of herpetic acute retinal necrosis and herpetic retrobulbar optic neuritis: five HIV-infected and one non-infected patients |journal=AJNR Am J Neuroradiol |volume=25 |issue=10 |pages=1722–9 |year=2004 |pmid=15569737 |doi= |url=}}</ref> | |||
*Increased T2 signal intensity in the optic pathway: [[optic nerves]], [[optic chiasm]], [[lateral geniculate bodies]], [[optic radiations]], [[visual cortex]], [[midbrain]] structures, [[trigeminal nerves]], and [[meninges]]. | |||
**The increased intensity reveals lesions that may be indicative of [[Herpes simplex virus]] or [[Cytomegalovirus]] infection. | |||
*[[Contrast-enhanced|Contrast enhanced CT]] T1-weighted images may reveal enhancement of [[optic nerve]], [[optic chiasm]], [[optic tracts]], [[optic radiation]], semilunar ganglion–Meckel cave, [[meninges]], and [[midbrain]]. | |||
==References== | ==References== |
Revision as of 17:22, 24 August 2016
Acute retinal necrosis Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Luke Rusowicz-Orazem, B.S.
Overview
Key MRI Findings for Acute retinal necrosis
MRI imaging may reveal the following indicators of Acute retinal necrosis:[1]
- Increased T2 signal intensity in the optic pathway: optic nerves, optic chiasm, lateral geniculate bodies, optic radiations, visual cortex, midbrain structures, trigeminal nerves, and meninges.
- The increased intensity reveals lesions that may be indicative of Herpes simplex virus or Cytomegalovirus infection.
- Contrast enhanced CT T1-weighted images may reveal enhancement of optic nerve, optic chiasm, optic tracts, optic radiation, semilunar ganglion–Meckel cave, meninges, and midbrain.