Acute retinal necrosis primary prevention: Difference between revisions

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Revision as of 15:24, 12 September 2016

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Luke Rusowicz-Orazem, B.S.

Preventing the onset of acute retinal necrosis is dependent upon prevention of the causative infection from Herpes simplex virus (HSV), Varicella-zoster virus (VZV), Cytomegalovirus (CMV), and Epstein-Barr virus (EBV).

Preventing the onset of acute retinal necrosis is dependent upon prevention of the causative infection from Herpes simplex virus (HSV), Varicella-zoster virus (VZV), and Cytomegalovirus (CMV). Measures to prevent viral infection include the following:^[1]^[2]^[3]

Avoiding oral and genital contact with individuals infected with HSV
Avoiding proximity to individuals infected with VZV to avoid contact with pathogenic respiratory droplets and fluid contact
Avoiding fluid contact with individuals infected with CMV or Epstein-Barr virus (EBV)

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 ==Overview==
-Preventing onset of acute [[retinal]] [[necrosis]] is dependent upon prevention of the causative infection from [[Herpes simplex virus]] (HSV), [[Varicella-zoster virus]] (VZV), [[Cytomegalovirus]] (CMV), and [[Epstein-Barr virus]] (EBV).
+Preventing the onset of acute [[retinal]] [[necrosis]] is dependent upon prevention of the causative infection from [[Herpes simplex virus]] (HSV), [[Varicella-zoster virus]] (VZV), [[Cytomegalovirus]] (CMV), and [[Epstein-Barr virus]] (EBV).
 ==Primary Prevention==