Postpartum thyroiditis pathophysiology: Difference between revisions
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{{CMG}}; {{AE}} | {{CMG}}; {{AE}} | ||
==Overview== | ==Overview== | ||
The exact pathogenesis of [ | The exact pathogenesis of [ Postpartum thyroiditis] is not fully understood. | ||
OR | OR | ||
It is thought that [ | It is thought that [ Postpartum thyroiditis] is mediated by either [Autoimmunity], [enviromental factors], or [smoking]. | ||
==Pathophysiology== | ==Pathophysiology== | ||
===Pathogenesis=== | ===Pathogenesis=== | ||
*The exact pathogenesis of postpartum thyroiditis is not fully understood | *The exact pathogenesis of postpartum thyroiditis is not fully understood. i | ||
OR | OR | ||
*It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | *It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | ||
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==Microscopic Pathology== | ==Microscopic Pathology== | ||
*On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | *On microscopic histopathological analysis it hs rer sermblance to Hishimoto thyriodtis but less degree of fibrosis and atrophy , [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | ||
==References== | ==References== |
Revision as of 20:23, 20 September 2017
Postpartum thyroiditis Microchapters |
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Postpartum thyroiditis pathophysiology On the Web |
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Risk calculators and risk factors for Postpartum thyroiditis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
The exact pathogenesis of [ Postpartum thyroiditis] is not fully understood.
OR
It is thought that [ Postpartum thyroiditis] is mediated by either [Autoimmunity], [enviromental factors], or [smoking].
Pathophysiology
Pathogenesis
- The exact pathogenesis of postpartum thyroiditis is not fully understood. i
OR
- It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the[histological subtype.
Genetics
- [Disease name] is transmitted in [mode of genetic transmission] pattern.
- Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
- The development of [disease name] is the result of multiple genetic mutations.
Associated Conditions
Gross Pathology
- On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
- On microscopic histopathological analysis it hs rer sermblance to Hishimoto thyriodtis but less degree of fibrosis and atrophy , [feature1], [feature2], and [feature3] are characteristic findings of [disease name].