Postpartum thyroiditis risk factors: Difference between revisions
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==Overview== | ==Overview== | ||
The most potent risk | The most potent risk factors in the development of [[Postpartum thyroiditis|postpartum thyroiditis PPT]] are [[Genetics|genetic]] abnormalities and subsequent pregnancies. Other risk factors for postpartum thyroiditis include [[smoking]], increase or decreased intake of [[Iodine]], [[hepatitis C]], radiations and [[medications]].<ref name="pmid25507668">{{cite journal| author=Argatska AB, Nonchev BI| title=Postpartum thyroiditis. | journal=Folia Med (Plovdiv) | year= 2014 | volume= 56 | issue= 3 | pages= 145-51 | pmid=25507668 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25507668 }}</ref> | ||
==Risk Factors== | ==Risk Factors== | ||
*The most potent risk | *The most potent [[Risk factor|risk factors]] in the development of [[Postpartum thyroiditis|PPT]] is [[Genetics|genetic]] abnormalities and multiple [[Pregnancy|pregnancies]]. Other risk factors include smoking, increase or decreased intake of [[Iodine]], [[hepatitis C]], [[Radiation exposure|radiations]] and certain medications. | ||
===Common Risk Factors=== | ===Common Risk Factors=== | ||
*Common risk | *Common risk factors in the development of [[Postpartum thyroiditis|PPT]] is [[Genetics|genetic]] abnormalities in: | ||
**CD60 CTLA-4 [[gene]] | **CD60 CTLA-4 [[gene]] | ||
**[[HLA DR3-DQ2|HLA DR3]] | **[[HLA DR3-DQ2|HLA DR3]] haplotype | ||
**[[HLA-DR4|HLA DR4]] | **[[HLA-DR4|HLA DR4]] haplotype | ||
**HLA [[HLA-DR5|DR5]] | **HLA [[HLA-DR5|DR5]] haplotype | ||
* Subsequent pregnancies are also a common risk factor for postpartum thyroiditis. | |||
===Less Common Risk Factors=== | ===Less Common Risk Factors=== | ||
*Less common risk factors in the development of [[Postpartum thyroiditis|PPT]] are environmental and they include: | *Less common risk factors in the development of postpartum thyroiditis ([[Postpartum thyroiditis|PPT]]) are environmental and they include: | ||
* | *#[[Smoking]] | ||
* | *#Intake of [[Iodine]] | ||
* | *#[[Radiation]] | ||
* | *#Medications: | ||
* | *#*[[Lithium]], [[amiodarone]], [[Interferon alpha|interferon alpha,]] [[interleukin 2]], and the highly active [[HIV AIDS medical therapy|anti-retro-viral therapy]]. | ||
* | |||
* | *#[[Systemic lupus erythematosus|Systemic lupus erythromatosis]] | ||
*#[[Multiple Sclerosis]] | |||
*#[[Thyroid]] hormone resistance syndrome | |||
==References== | ==References== |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sunny Kumar MD [2]
Overview
The most potent risk factors in the development of postpartum thyroiditis PPT are genetic abnormalities and subsequent pregnancies. Other risk factors for postpartum thyroiditis include smoking, increase or decreased intake of Iodine, hepatitis C, radiations and medications.[1]
Risk Factors
- The most potent risk factors in the development of PPT is genetic abnormalities and multiple pregnancies. Other risk factors include smoking, increase or decreased intake of Iodine, hepatitis C, radiations and certain medications.
Common Risk Factors
- Subsequent pregnancies are also a common risk factor for postpartum thyroiditis.
Less Common Risk Factors
- Less common risk factors in the development of postpartum thyroiditis (PPT) are environmental and they include:
- Smoking
- Intake of Iodine
- Radiation
- Medications:
- Lithium, amiodarone, interferon alpha, interleukin 2, and the highly active anti-retro-viral therapy.
- Systemic lupus erythromatosis
- Multiple Sclerosis
- Thyroid hormone resistance syndrome
References
- ↑ Argatska AB, Nonchev BI (2014). "Postpartum thyroiditis". Folia Med (Plovdiv). 56 (3): 145–51. PMID 25507668.