Rectal prolapse pathophysiology: Difference between revisions
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*Redundant sigmoid colon | *Redundant sigmoid colon | ||
*Patulous anal sphincter | *Patulous anal sphincter | ||
*Loss or attenuation of the rectal sacral attachments | *Loss or attenuation of the rectal sacral attachments<ref name="pmid28991074">{{cite journal |vauthors=Bordeianou L, Paquette I, Johnson E, Holubar SD, Gaertner W, Feingold DL, Steele SR |title=Clinical Practice Guidelines for the Treatment of Rectal Prolapse |journal=Dis. Colon Rectum |volume=60 |issue=11 |pages=1121–1131 |year=2017 |pmid=28991074 |doi=10.1097/DCR.0000000000000889 |url=}}</ref> | ||
==References== | ==References== |
Revision as of 17:15, 18 January 2018
Rectal prolapse Microchapters |
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Rectal prolapse pathophysiology On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: {{}}
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Pathogenesis
- The exact pathogenesis of [disease name] is not fully understood.
OR
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
- [Disease name] is transmitted in [mode of genetic transmission] pattern.
- Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
- The development of [disease name] is the result of multiple genetic mutations.
Associated Conditions
Rectal prolapse is associated with several coexisting anatomic abnormalities:
- Diastasis of the levator ani
- Abnormally deep cul-de-sac
- Redundant sigmoid colon
- Patulous anal sphincter
- Loss or attenuation of the rectal sacral attachments[1]
References
- ↑ Bordeianou L, Paquette I, Johnson E, Holubar SD, Gaertner W, Feingold DL, Steele SR (2017). "Clinical Practice Guidelines for the Treatment of Rectal Prolapse". Dis. Colon Rectum. 60 (11): 1121–1131. doi:10.1097/DCR.0000000000000889. PMID 28991074.