SLC25A38: Difference between revisions
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m Reverted 1 edit by 173.28.219.250 (talk) to last revision by Tyleroakley. (TW) |
imported>Jmertel23 Added {{unreferenced}} tag to article (TW) |
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{{protein | {{protein | ||
|name=solute carrier family 25, member 38 | |name=solute carrier family 25, member 38 | ||
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|LocusSupplementaryData= | |LocusSupplementaryData= | ||
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'''SLC25A38''' is a human gene. | '''SLC25A38''' is a human gene. SLC25A38 is involved in mitochondrial handling of glycine and is needed for the first step in heme synthesis. Mutations in this gene can lead to an autosomal recessive form of [[sideroblastic anemia]]. | ||
{{Membrane transport proteins}} | {{Membrane transport proteins}} | ||
{{gene-3-stub}} |
Latest revision as of 21:49, 5 July 2018
This article does not cite any sources. (July 2018) (Learn how and when to remove this template message) |
solute carrier family 25, member 38 | |
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Identifiers | |
Symbol | SLC25A38 |
Entrez | 54977 |
HUGO | 26054 |
OMIM | 610819 |
RefSeq | NM_017875 |
Other data | |
Locus | Chr. 3 p22.1 |
SLC25A38 is a human gene. SLC25A38 is involved in mitochondrial handling of glycine and is needed for the first step in heme synthesis. Mutations in this gene can lead to an autosomal recessive form of sideroblastic anemia.
This article on a gene on human chromosome 3 is a stub. You can help Wikipedia by expanding it. |