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{{Dextro-transposition of the great arteries/complete transposition of the great arteries}}
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{{Transposition of the great vessels}}


{{CMG}}; '''Associate Editor(s)-In-Chief:''' [[Priyamvada Singh|Priyamvada Singh, M.B.B.S.]] [mailto:psingh@perfuse.org]; {{CZ}}; [[User:KeriShafer|Keri Shafer, M.D.]] [mailto:kshafer@bidmc.harvard.edu]; '''Assistant Editor(s)-In-Chief:''' [[Kristin Feeney|Kristin Feeney, B.S.]] [mailto:kfeeney@perfuse.org]


{{CMG}}
==Overview==
 
'''Associate Editors-In-Chief:''' [[User:KeriShafer|Keri Shafer, M.D.]] [mailto:kshafer@bidmc.harvard.edu]; Atif Mohammad, M.D., [[Priyamvada Singh]], [[MBBS]]
 
 
==== Physical Examination in Infants with d-TGA ====


==Physical Examination in Infants with d-TGA==
Symmetric cyanosis is the main characteristic in physical appearence of patients with d-TGA, inadequate mixing, and low pulmonary arterial blood flow. Delayed mild cyanosis, and the apearence of congestive heart failure accompanies non-restrictive VSD.  
Symmetric cyanosis is the main characteristic in physical appearence of patients with d-TGA, inadequate mixing, and low pulmonary arterial blood flow. Delayed mild cyanosis, and the apearence of congestive heart failure accompanies non-restrictive VSD.  



Revision as of 03:48, 14 August 2011

Dextro-transposition of the great arteries Microchapters

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Patient Information

Overview

Classification

Pathophysiology

Causes

Differentiating dextro-transposition of the great arteries from other Diseases

Epidemiology and Demographics

Screening

Pre-natal dextro-transposition of the great arteries
Post-natal dextro-transposition of the great arteries
Infants with dextro-transposition of the great arteries

Natural History, Complications and Prognosis

Diagnosis

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

Chest X Ray

Echocardiography or Ultrasound

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Transposition of the great vessels Microchapters

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Patient Information

Overview

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Classification

Dextro-transposition of the great arteries
L-transposition of the great arteries

Pathophysiology

Causes

Differentiating Transposition of the great vessels from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

Chest X Ray

MRI

CT

Echocardiography

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Dextro-transposition of the great arteries physical examination On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Dextro-transposition of the great arteries physical examination

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Dextro-transposition of the great arteries physical examination

CDC on Dextro-transposition of the great arteries physical examination

Dextro-transposition of the great arteries physical examination in the news

Blogs on Dextro-transposition of the great arteries physical examination

Directions to Hospitals Treating Type page name here

Risk calculators and risk factors for Dextro-transposition of the great arteries physical examination

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.B.B.S. [2]; Cafer Zorkun, M.D., Ph.D. [3]; Keri Shafer, M.D. [4]; Assistant Editor(s)-In-Chief: Kristin Feeney, B.S. [5]

Overview

Physical Examination in Infants with d-TGA

Symmetric cyanosis is the main characteristic in physical appearence of patients with d-TGA, inadequate mixing, and low pulmonary arterial blood flow. Delayed mild cyanosis, and the apearence of congestive heart failure accompanies non-restrictive VSD.

Reversed differential cyanosis (feet less cyanotic than hands) can be a manifestation in patients with d-TGA and large patent ductus arteriosus that has develop early pulmonary vascular disease, reversing the ductal flow if the patient has survived. At this poin in time, the pulmonary arterial blood of high oxygen content enters the aorta and is selectively distribuited to the lower extremities.

The murmur of a large PDA in d-TGA, is usually systolic, seldom continuos, due to the almost exclusive flow during systole from the aorta to the pulmonary artery.

There is a prominent impulse at the LLSB (the RV which is actually the morphologic LV).

The first heart sound (S1) is normal in intensity and splitting because the PR interval and ventricular activation is normal. Due to the anterior location of the aorta, the second heart sound (S2) is accentuated and is usually single.

Systolic murmus are absent in neonates unless a subpulmonic stenosis is present. Short midsystolic murmur originate in the anterior aorta when hypervolemia is present. When the pulmonary vascular resistence is low, a midsystolic murmur is originated in the posterior pulmonary artery, but the murmur is dump by the anterior aorta.

A VSD murmur (holosystolic)is absent at birth, until the pulmonary vascular resistence fall. A subsequent increase in pulmonary resistence shortens and later abolishes the murmur.

References

Acknowledgements and Initial Contributors to Page

Leida Perez, M.D.

External links

nl:Transpositie van de grote vaten

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